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J Am Acad Orthop Surg, Vol 14, No 5, May 2006, 303-311.
© 2006 the American Academy of Orthopaedic Surgeons
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Renal Osteodystrophy

Nirmal C. Tejwani, MD, Aaron K. Schachter, MD, Igor Immerman, BS and Pramod Achan, MBBS, FRCS (Orth)

Dr. Tejwani is Associate Professor, Department of Orthopaedics, Bellevue Hospital, NYU–Hospital for Joint Diseases, New York, NY. Dr. Schachter is Resident, NYU–Hospital for Joint Diseases. Mr. Immerman is a Medical Student, NYU–Hospital for Joint Diseases. Dr. Achan is Fellow, Department of Orthopaedics, NYU–Hospital for Joint Diseases.

None of the following authors or the departments with which they are affiliated has received anything of value from or owns stock in a commercial company or institution related directly or indirectly to the subject of this article: Dr. Tejwani, Dr. Schachter, Mr. Immerman, and Dr. Achan.

Reprint requests: Dr. Tejwani, Bellevue Hospital, 550 First Avenue, NBV 21W37, New York, NY 10016.

The incidence of chronic renal disease is increasing, and the pattern of renal osteodystrophy seems to be shifting from the classic hyperparathyroid presentation to one of low bone turnover. Patients with persistent disease also live longer than previously and are more physically active. Thus, patients may experience trauma as a direct result of increased physical activity in a setting of weakened pathologic bone. Patient quality of life is primarily limited by musculoskeletal problems, such as bone pain, muscle weakness, growth retardation, and skeletal deformity. Chronic renal disease also increases the risk of comorbidity, such as infection, bleeding, and anesthesia-related problems. Current treatment strategies include dietary changes, plate-and-screw fixation, and open reduction and internal fixation.






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Copyright © 2006 by the American Academy of Orthopaedic Surgeons.